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Dr, Jesús Porta-Etessam

Servicio de Neurología
Universidad Complutense
Hospital Clínico San Carlos de 
Madrid

Instituto de Neurociencias Aplicadas

C/ La granja, 8. Madrid

Consulta 5.5

Citaciones: 91 299 12 99

Redes sociales :

Noticias destacadas

Neurología

Evolución a largo plazo de la hidrocefalia crónica del adulto idiopática tratada con válvula de derivación ventrículo-peritoneal
I. Illán-Gala, J. Pérez-Lucas, A. Martín-Montes, J. Máñez-Miró, J. Arpa, G. Ruiz-Ares Neurologia 2017;32:205-12 Resumen - Texto completo - PDF


Hemorragia subaracnoidea espontánea de la convexidad cerebral: una serie clínica de 3 pacientes asociada con angiopatía amiloide cerebral
D.A. García Estévez, R.M. García-Dorrego, B. Nieto-Baltar, M. Marey-Garrido, T. Hierro-Torner Neurologia 2017;32:213-8 Resumen - Texto completo - PDF


Beyond clinical syndromes in primary progressive aphasia: Seeking etiologic diagnoses
Primary progressive aphasia (PPA) is an acquired disorder in which speech and language problems are the dominant (salient) manifestations during the initial symptomatic illness.1 Because of substantial syndromic–etiologic heterogeneity in some subtypes of PPA, it is a case study in the challenges of linking clinical syndromes to etiology. In a disorder like PPA, there is a pressing need to develop methodologies to diagnose underlying etiology. While definitive treatment for the neurodegenerative cognitive disorders is still in the future, basic and translational neuroscience is at the threshold of testing promising new therapies. Precise diagnosis of underlying brain pathophysiology is integral to success in clinical trials. Development of PET imaging biomarkers has great potential for offering direct confirmation of etiology, but PET imaging is expensive. The article by Giannini et al.2 in this issue of Neurology® applies the low-technology clinical observation approach for seeking indicators of PPA etiologic subtypes.
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Thalamus as a "hub" to predict outcome after epilepsy surgery
The role of the thalamus in mediating seizure activity has been investigated for decades.1 In 1952, Penfield2 proposed "centrencephalic" interactions as an integral part not only of generalized but also of focal automatisms. The dynamic integration between the centrencephalic system and cerebral cortex was hypothesized as pivotal for normal brain function, as well as a mechanism for seizure propagation. The concept was later hotly debated, and generalized seizures with 3-Hz spike wave discharges were considered mainly centrencephalic.1
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Dr Jesús Porta-Etessam. Madrid